Synaptotagmin I- and II-deficient PC12 cells exhibit calcium-independent, depolarization-induced neurotransmitter release from synaptic-like microvesicles

FEBS Letters

Volumn 364, Issue 3, 1995, Pages 328-334

  Bauerfeind, Rudolf ^a   Jelinek, Ruth ^a   Huttner, Wieland B ^a  

^a UNIVERSITY OF HEIDELBERG   (Germany)

Author keywords

Calcium; Exocytosis; Regulated secretion; Secretory granule; Synaptic vesicle; Synaptotagmin

Indexed keywords

CALCIUM; SYNAPTOTAGMIN;

ANIMAL CELL; ARTICLE; CALCIUM TRANSPORT; EXOCYTOSIS; NEUROTRANSMITTER RELEASE; NONHUMAN; PRIORITY JOURNAL; RAT; SECRETORY GRANULE; SYNAPSE VESICLE;

ACETYLCHOLINE; ACTION POTENTIALS; ANIMAL; CALCIUM; CENTRIFUGATION, DENSITY GRADIENT; CHOLINE; CYTOPLASMIC GRANULES; EXOCYTOSIS; MEMBRANE GLYCOPROTEINS; NERVE TISSUE PROTEINS; NEUROTRANSMITTERS; PC12 CELLS; RATS; SUPPORT, NON-U.S. GOV'T; SYNAPTIC VESICLES; TRITIUM;

EID: 0029037547     PISSN: 00145793     EISSN: None     Source Type: Journal    
DOI: 10.1016/0014-5793(95)00419-A     Document Type: Article

References (62)

1. Absence of synaptotagmin disrupts excitation-secretion coupling during synaptic transmission.
2. Genetic analysis of neurotransmitter release at the synapse