Signaling pathways and c-fos transcriptional response — links to inherited diseases

New England Journal of Medicine

Volumn 332, Issue 23, 1995, Pages 1576-1577

  Sassone Corsi, Paolo ^a  

^a INSTITUT DE GÉNÉTIQUE ET DE BIOLOGIE MOLÉCULAIRE ET CELLULAIRE   (France)

Author keywords

[No Author keywords available]

Indexed keywords

TRANSCRIPTION FACTOR;

BONE BIOPSY; CYCLIC AMP RESPONSIVE ELEMENT; EDITORIAL; FIBROUS DYSPLASIA; GENE EXPRESSION; GENETIC TRANSCRIPTION; HUMAN; ONCOGENE C FOS; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

CYCLIC AMP; FIBROUS DYSPLASIA OF BONE; GENE EXPRESSION REGULATION; GENES, FOS; GTP-BINDING PROTEINS; HUMAN; PROTO-ONCOGENE PROTEINS C-JUN; SIGNAL TRANSDUCTION;

EID: 0029024661     PISSN: 00284793     EISSN: 15334406     Source Type: Journal    
DOI: 10.1056/NEJM199506083322311     Document Type: Editorial

References (6)

1. Sassone-Corsi P, Borrelli E Mutations in signal transduction pathways and inherited diseases. Curr Opin Genet Dev 1992; 2: 455-458
2. Lewin B Oncogenic conversion by regulatory changes in transcription factors. Cell 1991; 64: 303-312
3. Candeliere GA, Glorieux FH, Prud'homme J, St Arnaud R Increased expression of the c-fos proto-oncogene in bone from patients with fibrous dysplasia. N Engl J Med 1995; 332: 1546-1551
4. Lalli E, Sassone-Corsi P Signal transduction and gene regulation: the nuclear response to cAMP. J Biol Chem 1994; 269: 17359-17362
5. Angel P, Karin M The role of Jun, Fos and the AP-1 complex in cell-proliferation and transformation. Biochim Biophys Acta 1991; 1072: 129-157
6. Wong YH, Federman A, Pace AM, Mutant alpha subunits of Gi2 inhibit cyclic AMP accumulation. Nature 1991; 351: 63-65