TNFα receptor expression in rat cardiac myocytes: TNFα inhibition of L-type Ca2+ current and Ca2+ transients

FEBS Letters

Volumn 376, Issue 1-2, 1995, Pages 24-30

  Krown, Kevin A ^a   Yasui, Kenji ^b   Brooker, Madelyne J ^a   Dubin, Adrienne E ^a   Nguyen, Cuong ^a   Harris, Greg L ^a   McDonough, Patrick M ^a   Glembotski, Christopher C ^a   Palade, Philip T ^b   Sabbadini, Roger A ^a  

^a SAN DIEGO STATE UNIVERSITY   (United States)

^b UNIVERSITY OF TEXAS MEDICAL BRANCH   (United States)

Author keywords

Cardiac Ca2+; Inotropy; Rat cardiac myocyte; Single cell RT PCR; TNF receptor

Indexed keywords

TUMOR NECROSIS FACTOR ALPHA; TUMOR NECROSIS FACTOR RECEPTOR;

ANIMAL CELL; ARTICLE; CALCIUM CURRENT; CARDIOTOXICITY; CONTROLLED STUDY; HEART; INOTROPISM; NONHUMAN; PRIORITY JOURNAL; RAT;

ANIMAL; BASE SEQUENCE; CALCIUM; CALCIUM CHANNELS; DNA PRIMERS; FLUORESCENT DYES; GENE EXPRESSION; IMMUNOBLOTTING; IN VITRO; INDOLES; MOLECULAR SEQUENCE DATA; MYOCARDIUM; PATCH-CLAMP TECHNIQUES; POLYMERASE CHAIN REACTION; RATS; RATS, SPRAGUE-DAWLEY; RECEPTORS, TUMOR NECROSIS FACTOR; RNA, MESSENGER; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.; TUMOR NECROSIS FACTOR;

EID: 0028971714     PISSN: 00145793     EISSN: None     Source Type: Journal    
DOI: 10.1016/0014-5793(95)01238-5     Document Type: Article

References (51)

1. Elevated Circulating Levels of Tumor Necrosis Factor in Severe Chronic Heart Failure
2. Negative control mechanism with features of adaptation controls Ca2+ release in cardiac myocytes.