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Volumn 186, Issue 2-3, 1995, Pages 161-164
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Chronically administered 3-nitropropionic acid induces striatal lesions attributed to dysfunction of the blood-brain barrier
a
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Author keywords
3 nitropropionic acid; Blood brain barrier; Complement factor C3b C4br; GFAP; IgG; Neuronal death; Striatum
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Indexed keywords
3 NITROPROPIONIC ACID;
ENDOGENOUS COMPOUND;
ENZYME INHIBITOR;
GLIAL FIBRILLARY ACIDIC PROTEIN;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
BLOOD BRAIN BARRIER;
BRAIN INJURY;
CELL DEATH;
COMPLEMENT FACTOR;
CONTROLLED STUDY;
CORPUS STRIATUM;
IMMUNOCYTOCHEMISTRY;
MALE;
NONHUMAN;
PRIORITY JOURNAL;
RAT;
SUBCUTANEOUS DRUG ADMINISTRATION;
ANIMAL;
ANTIHYPERTENSIVE AGENTS;
BLOOD-BRAIN BARRIER;
BRAIN DISEASES;
COMPLEMENT 3B;
COMPLEMENT 4B;
GLIAL FIBRILLARY ACIDIC PROTEIN;
IMMUNOGLOBULIN G;
IMMUNOHISTOCHEMISTRY;
MALE;
NEOSTRIATUM;
NERVE DEGENERATION;
PROPIONIC ACIDS;
RATS;
RATS, WISTAR;
SUPPORT, NON-U.S. GOV'T;
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EID: 0028942180
PISSN: 03043940
EISSN: None
Source Type: Journal
DOI: 10.1016/0304-3940(95)11311-J Document Type: Article |
Times cited : (62)
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References (17)
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