Mice deficient in cellular retinoic acid binding protein II (CRABPII) or in both CRABPI and CRABPII are essentially normal

Development

Volumn 121, Issue 2, 1995, Pages 539-548

  Lampron, C ^a   Rochette Egly, C ^a   Gorry, P ^a   Dolle, P ^a   Mark, M ^a   Lufkin, T ^a   LeMeur, M ^a   Chambon, P ^a  

^a INSERM   (France)

Author keywords

CRABPI and CRABPII double mutant; CRABPII mutant; Gene targeting; Mouse development; Retinoic acid signaling pathway; Vitamin A

Indexed keywords

RETINOIC ACID; RETINOIC ACID BINDING PROTEIN;

ANIMAL BEHAVIOR; ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; CONTROLLED STUDY; EMBRYO; EMBRYO CELL; EVOLUTION; FEMALE; FERTILITY; GENE DISRUPTION; GENE MUTATION; GENETIC CONSERVATION; GENETIC RECOMBINATION; LIFESPAN; MALE; MOUSE; NONHUMAN; POSTNATAL DEVELOPMENT; PRENATAL DEVELOPMENT; PRIORITY JOURNAL; STEM CELL;

ANIMAL; EXTREMITIES; LIMB DEFORMITIES, CONGENITAL; MICE; MICE, MUTANT STRAINS; MORPHOGENESIS; RECEPTORS, RETINOIC ACID; SUPPORT, NON-U.S. GOV'T; TRETINOIN;

EID: 0028912640     PISSN: 09501991     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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