Mycobacterium tuberculosis is a natural mutant with an inactivated oxidative‐stress regulatory gene:implications for sensitivity to isoniazid

Molecular Microbiology

Volumn 17, Issue 5, 1995, Pages 889-900

  Deretic, V ^a   Philipp, W ^b   Dhandayuthapani, S ^a   Mudd, M H ^a   Curcic, R ^a   Garbe, T ^a   Heym, B ^b   Via, L E ^a   Cole, S T ^b  

^a UNIVERSITY OF TEXAS HEALTH SCIENCE CENTER AT SAN ANTONIO   (United States)

^b INSTITUT PASTEUR   (France)

Author keywords

[No Author keywords available]

Indexed keywords

ISONIAZID;

ANTIBIOTIC RESISTANCE; ARTICLE; BACTERIUM MUTANT; CONTROLLED STUDY; GENE CONTROL; MYCOBACTERIUM TUBERCULOSIS; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL;

AMINO ACID SEQUENCE; BACTERIAL PROTEINS; BASE SEQUENCE; CATALASE; COMPARATIVE STUDY; GENES, BACTERIAL; GENES, REGULATOR; HUMAN; ISONIAZID; MOLECULAR SEQUENCE DATA; MUTATION; MYCOBACTERIUM; MYCOBACTERIUM BOVIS; MYCOBACTERIUM LEPRAE; MYCOBACTERIUM TUBERCULOSIS; OXIDATIVE STRESS; OXIDOREDUCTASES; PEROXIDASES; REPRESSOR PROTEINS; SEQUENCE HOMOLOGY, AMINO ACID; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.; TRANSCRIPTION FACTORS;

EID: 0028882958     PISSN: 0950382X     EISSN: 13652958     Source Type: Journal    
DOI: 10.1111/j.1365-2958.1995.mmi_17050889.x     Document Type: Article

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