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Volumn 147, Issue 5, 1995, Pages 1193-1199
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Suppression of insulitis in non-obese diabetic (NOD) mice by oral insulin administration is associated with selective expression of interleukin-4 and - 10, transforming growth factor-β, and prostaglandin-E
a
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Author keywords
[No Author keywords available]
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Indexed keywords
AUTOANTIGEN;
CD4 ANTIGEN;
GAMMA INTERFERON;
INSULIN;
INTERLEUKIN 10;
INTERLEUKIN 2;
INTERLEUKIN 4;
OVALBUMIN;
PROSTAGLANDIN E;
TRANSFORMING GROWTH FACTOR BETA;
TUMOR NECROSIS FACTOR ALPHA;
ADOPTIVE TRANSFER;
ANIMAL CELL;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
CONTROLLED STUDY;
DIABETES MELLITUS;
DIABETOGENESIS;
FEMALE;
GENE EXPRESSION REGULATION;
HISTOPATHOLOGY;
IMMUNOMODULATION;
INSULIN TREATMENT;
INSULITIS;
LYMPHOCYTIC INFILTRATION;
MOUSE;
NONHUMAN;
ORAL DRUG ADMINISTRATION;
PRIORITY JOURNAL;
SUPPRESSOR GENE;
ADMINISTRATION, ORAL;
ANIMAL;
DIABETES MELLITUS, INSULIN-DEPENDENT;
FEMALE;
INSULIN;
INTERLEUKIN-10;
INTERLEUKIN-4;
ISLETS OF LANGERHANS;
MICE;
MICE, INBRED NOD;
PROSTAGLANDINS E;
SUPPORT, U.S. GOV'T, P.H.S.;
TRANSFORMING GROWTH FACTOR BETA;
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EID: 0028882673
PISSN: 00029440
EISSN: None
Source Type: Journal
DOI: None Document Type: Article |
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Times cited : (175)
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References (0)
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