Two closely related isoforms of protein kinase C produce reciprocal effects on the growth of rat fibroblasts. Possible molecular mechanisms

Journal of Biological Chemistry

Volumn 270, Issue 1, 1995, Pages 78-86

  Borner, C ^a   Ueffing, M ^a   Jaken, S ^a   Parker, P J ^a   Weinstein, I B ^a  

^a Howard Hughes Medical Institute   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

PROTEIN KINASE C;

ANCHORAGE INDEPENDENT GROWTH; ANIMAL CELL; ARTICLE; CANCER; CARCINOGENESIS; CELL GROWTH; CELL TRANSFORMATION; ENZYME ACTIVITY; NONHUMAN; PRIORITY JOURNAL; RAT; SIGNAL TRANSDUCTION; TUMOR; TUMOR GROWTH;

ANIMAL; BLOOD; CELL DIVISION; CELL LINE; DOWN-REGULATION; ENZYME ACTIVATION; FIBROBLASTS; GENES, IMMEDIATE-EARLY; GROWTH SUBSTANCES; ISOENZYMES; MICE; MICE, NUDE; PROTEIN KINASE C; RATS; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.;

EID: 0028815491     PISSN: 00219258     EISSN: None     Source Type: Journal    
DOI: 10.1074/jbc.270.1.78     Document Type: Article

References (0)