World distribution of factor V Leiden

The Lancet

Volumn 346, Issue 8983, 1995, Pages 1133-1134

  Rees, D C ^a   Cox, M ^a   Clegg, J B ^a  

^a JOHN RADCLIFFE HOSPITAL   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

ANTITHROMBIN III; BLOOD CLOTTING FACTOR 5; HEPARIN COFACTOR II; PROTEIN C; PROTEIN S;

AFRICA; ARTICLE; ASIA; AUSTRALIA; CHROMOSOME ANALYSIS; ETHNIC GROUP; GENE FREQUENCY; GREECE; HUMAN; POINT MUTATION; POLYMERASE CHAIN REACTION; POPULATION RESEARCH; PRIORITY JOURNAL; RISK FACTOR; SOUTHEAST ASIA; THROMBOPHILIA; VEIN THROMBOSIS; WESTERN HEMISPHERE;

EID: 0028810738     PISSN: 01406736     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0140-6736(95)91803-5     Document Type: Article

References (10)

1. The molecular genetics of familial venous thombosis
2. Familial thrombophilia due to a previously unrecognised mechanism characterized by poor anticoagulant response to activated protein C: prediction of a cofactor to activated protein C
3. Venous thrombosis due to poor anticoagulant response to activated protein C: Leiden Thrombophilia Study
4. Mutation in blood coagulation factor V associated with resistance to activated protein C
5. High risk of thrombosis in patients homozygous for factor V Leiden (activated protein C resistance)
6. Activated protein C resistance as an additional risk factor for thrombosis in protein C-deficient families
7. Mutation in the gene coding for coagulation factor V and the risk of myocardial infarction, stroke and venous thrombosis in apparently healthy men
8. Why are some genetic diseases common? Distinguishing selection from other processes by molecular analysis of globin gene variants
9. Varicose veins, deep vein thrombosis, and haemorrhoids: epidemiology and suggested aetiology
10. Epidemiology of coagulation disorders