Testing the role of p53 in the expression of genetic instability and apoptosis in ataxia-telangiectasia

International Journal of Radiation Biology

Volumn 66, Issue 6, 1994, Pages

  Meyn, M S ^a   Strasfeld, L ^a   Allen, C ^a  

^a YALE UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

APOPTOSIS; ATAXIA TELANGIECTASIA; CONFERENCE PAPER; CONTROLLED STUDY; GENE FUNCTION; HUMAN; HUMAN CELL; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

APOPTOSIS; ATAXIA TELANGIECTASIA; DNA; FIBROBLASTS; GENES, P53; HUMAN; MUTATION; ONCOGENE PROTEINS, VIRAL; PHENOTYPE; PROTEIN P53; RECOMBINATION, GENETIC; STREPTONIGRIN; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.; TRANSFECTION;

EID: 0028566172     PISSN: 09553002     EISSN: None     Source Type: Journal    
DOI: 10.1080/09553009414551971     Document Type: Article

References (29)

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14. Wild-type p53 is a cell cycle checkpoint determinant following irradiation
15. p53 mutations increase resistance to ionizing radiation
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17. Altered cell cycle arrest and gene amplification potential accompany loss of wild-type p53
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23. Ataxia-Telangiectasia
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