Neurotrophins: Keeping track of changing neurotrophic theory

Current Biology

Volumn 4, Issue 7, 1994, Pages 662-665

  Johnson, James ^a   Oppenheim, Ronald ^a  

^a WAKE FOREST SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

NERVE GROWTH FACTOR; NERVE GROWTH FACTOR RECEPTOR;

ANIMAL; APOPTOSIS; BIOLOGICAL MODEL; CENTRAL NERVOUS SYSTEM; CHEMICAL STRUCTURE; CHEMISTRY; CONGENITAL MALFORMATION; CYTOLOGY; EMBRYOLOGY; GENETICS; HISTOLOGY; LETHAL GENE; MOUSE; MOUSE MUTANT; NERVE CELL PLASTICITY; PHENOTYPE; PHYSIOLOGY; PRENATAL DEVELOPMENT; REVIEW; STRUCTURAL GENE; TRANSGENIC MOUSE;

ANIMAL; APOPTOSIS; CENTRAL NERVOUS SYSTEM; EMBRYO AND FETAL DEVELOPMENT; GENES, LETHAL; GENES, STRUCTURAL; MICE; MICE, KNOCKOUT; MICE, NEUROLOGIC MUTANTS; MICE, TRANSGENIC; MODELS, MOLECULAR; MODELS, NEUROLOGICAL; NERVE GROWTH FACTORS; NEURONAL PLASTICITY; PHENOTYPE; RECEPTORS, NERVE GROWTH FACTOR;

ANIMALIA; MUS MUSCULUS;

EID: 0028467370     PISSN: 09609822     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0960-9822(00)00149-4     Document Type: Article

References (16)

1. Cell death in the nervous system
2. The 1986 Nobel Prize in physiology or medicine
3. New neurotrophic factors
4. The changing scene of neurotrophic factors
5. Tracking neurotrophin function
6. Severe sensory and sympathetic neuropathies in mice carrying a disrupted trk/NGF receptor gene
7. Targeted disruption of the trkB neurotrophin receptor gene results in nervous system lesions and neonatal death
8. Disruption of the neurotrophin 3 receptor gene trkC eliminates Ia muscle afferents and results in abnormal movements
9. Mice lacking brain-derived neurotrophic factor develop with sensory deficits
10. Targeted disruption to the BDNF gene perturbs brain and sensory neuron development but not motor neuron development
11. Lack of neurotrophin 3 leads to deficiencies in the peripheral nervous system and loss of limb proprioceptive afferents
12. Targeted disruption of the c-src proto-oncogene leads to osteoporosis in mice
13. Targeted disruption of the mouse transforming growth factor β-1 gene results in multifocal inflammatory disease
14. Mice develop normally without tenascin
15. Short-term synaptic plasticity is altered in mice lacking synapsin
16. Inactivation of MyoD in mice leads to up-regulation of the myogenic HLH gene Myf-5 and results in apparently normal muscle development