A role for non-MHC genetic polymorphism in susceptibility to spontaneous autoimmunity

Immunity

Volumn 1, Issue 1, 1994, Pages 73-82

  Scott, Bernadette ^a   Liblau, Roland ^b   Degermann, Sylvia ^a   Marconi, Lori Anne ^a   Ogata, Lynn ^a   Caton, Andrew J ^c   McDevitt, Hugh O ^b   Lo, David ^a  

^a SCRIPPS RESEARCH INSTITUTE   (United States)

^b STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c WISTAR INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CYTOKINE; LYMPHOCYTE ANTIGEN RECEPTOR; RECEPTORS, ANTIGEN, T CELL; VIRUS HEMAGGLUTININ;

ANIMAL; ARTICLE; AUTOIMMUNITY; BIOSYNTHESIS; EXPERIMENTAL DIABETES MELLITUS; FEMALE; GENETIC POLYMORPHISM; GENETICS; IMMUNOLOGICAL MEMORY; IMMUNOLOGICAL TOLERANCE; IMMUNOLOGY; LYMPHOCYTE ACTIVATION; MAJOR HISTOCOMPATIBILITY COMPLEX; MALE; MOUSE; MOUSE STRAIN; ORTHOMYXOVIRUS; PANCREAS ISLET; PATHOLOGY; PEDIGREE; T LYMPHOCYTE; TRANSGENIC MOUSE;

ANIMAL; AUTOIMMUNITY; CD4-POSITIVE T-LYMPHOCYTES; CYTOKINES; DIABETES MELLITUS, EXPERIMENTAL; FEMALE; HEMAGGLUTININS, VIRAL; IMMUNE TOLERANCE; IMMUNOLOGIC MEMORY; ISLETS OF LANGERHANS; LYMPHOCYTE ACTIVATION; MAJOR HISTOCOMPATIBILITY COMPLEX; MALE; MICE; MICE, INBRED BALB C; MICE, TRANSGENIC; ORTHOMYXOVIRIDAE; PEDIGREE; POLYMORPHISM (GENETICS); RECEPTORS, ANTIGEN, T-CELL; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.;

EID: 0028409436     PISSN: 10747613     EISSN: None     Source Type: Journal    
DOI: 10.1016/1074-7613(94)90011-6     Document Type: Article

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