Abrogation of oncogene-associated apoptosis allows transformation of p53- deficient cells

Proceedings of the National Academy of Sciences of the United States of America

Volumn 91, Issue 6, 1994, Pages 2026-2030

  Lowe, S W ^a   Jacks, T ^a   Housman, D E ^a   Ruley, H E ^a  

^a VANDERBILT UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

adenovirus E1A; adenovirus E1B; tumor suppressor gene

Indexed keywords

HYGROMYCIN; PHOSPHOTRANSFERASE; PROTEIN P53; VIRUS PROTEIN;

ADENOVIRUS; ANIMAL CELL; ANIMAL EXPERIMENT; APOPTOSIS; ARTICLE; CARCINOGENICITY; CELL CYCLE; CELL PROLIFERATION; CELL SURVIVAL; CELL TRANSFORMATION; CONTROLLED STUDY; EMBRYO; FIBROBLAST CULTURE; GENE EXPRESSION; MALE; MOUSE; NONHUMAN; ONCOGENE RAS; PRIORITY JOURNAL; TUMOR GROWTH;

ADENOVIRIDAE; ANIMALIA;

EID: 0028323294     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.91.6.2026     Document Type: Article

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