Ablation of E2A in recombinant adenoviruses improves transgene persistence and decreases inflammatory response in mouse liver

Proceedings of the National Academy of Sciences of the United States of America

Volumn 91, Issue 13, 1994, Pages 6196-6200

  Engelhardt, John F ^a   Ye, Xuehai ^a   Doranz, Benjamin ^a   Wilson, James M ^a  

^a HOSPITAL OF THE UNIVERSITY OF PENNSYLVANIA   (United States)

Author keywords

temperature sensitive

Indexed keywords

CD8 ANTIGEN;

ADENOVIRUS; ANIMAL CELL; ARTICLE; CONTROLLED STUDY; GENE THERAPY; INFLAMMATION; LIVER; MOUSE; NONHUMAN; PRIORITY JOURNAL; T LYMPHOCYTE; TEMPERATURE SENSITIVE MUTANT; TRANSGENE; VIRUS RECOMBINANT;

ADENOVIRIDAE; ADENOVIRUS E2 PROTEINS; ANIMAL; ANTIGENS, CD8; BETA-GALACTOSIDASE; COMPARATIVE STUDY; CYCLOSPORINE; ESCHERICHIA COLI; GENE DELETION; GENE EXPRESSION; GENE THERAPY; GENES, VIRAL; GENETIC VECTORS; HUMAN; IMMUNOHISTOCHEMISTRY; INFLAMMATION; LIVER; MICE; MICE, TRANSGENIC; SUPPORT, U.S. GOV'T, P.H.S.;

EID: 0028246338     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.91.13.6196     Document Type: Article

References (14)