Evidence that BCL-2 represses apoptosis by regulating endoplasmic reticulum-associated Ca2+ fluxes

Proceedings of the National Academy of Sciences of the United States of America

Volumn 91, Issue 14, 1994, Pages 6569-6573

  Lam, M ^a   Dubyak, G ^a   Chen, L ^a   Nunez, G ^a   Miesfeld, R L ^a   Distelhorst, C W ^a  

^a CASE WESTERN RESERVE UNIVERSITY   (United States)

Author keywords

calcium homeostasis; glucocorticosteroid; mouse lymphoma cell; programmed cell death; thapsigargin

Indexed keywords

CALCIUM; DEXAMETHASONE; THAPSIGARGIN;

ANIMAL CELL; APOPTOSIS; ARTICLE; CALCIUM CELL LEVEL; CALCIUM HOMEOSTASIS; CALCIUM TRANSPORT; ENDOPLASMIC RETICULUM; EXTRACELLULAR CALCIUM; GENE EXPRESSION REGULATION; INTRACELLULAR TRANSPORT; LYMPHOMA CELL LINE; MOUSE; NONHUMAN; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

APOPTOSIS; CA(2+)-TRANSPORTING ATPASE; CALCIUM; CELL LINE; CELL MEMBRANE PERMEABILITY; CELL SURVIVAL; CLONE CELLS; COMPARATIVE STUDY; DEXAMETHASONE; DIGITONIN; ENDOPLASMIC RETICULUM; GTP-BINDING PROTEINS; HUMAN; KINETICS; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2; PROTO-ONCOGENES; RNA, MESSENGER; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.; TERPENES; THAPSIGARGIN; TIME FACTORS; TRANSFECTION;

EID: 0028216444     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.91.14.6569     Document Type: Article

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