Contribution of complement‐stimulated hepatic macrophages and neutrophils to endotoxin‐induced liver injury in rats

Hepatology

Volumn 19, Issue 4, 1994, Pages 973-979

  Jaeschke, Hartmut ^a,d   Farhood, Anwar ^c   Smith, C Wayne ^b  

^a Center for Experimental Therapeutics   (United States)

^b BAYLOR COLLEGE OF MEDICINE   (United States)

^c UNIVERSITY OF TEXAS   (United States)

^d PFIZER INC   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CD11B ANTIGEN; CD18 ANTIGEN; COMPLEMENT; CORYNEBACTERIUM PARVUM EXTRACT; GLUTATHIONE DISULFIDE; MONOCLONAL ANTIBODY; SALMONELLA ENDOTOXIN; TUMOR NECROSIS FACTOR ALPHA;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CELL INFILTRATION; COMPLEMENT ACTIVATION; IMMUNE MEDIATED INJURY; LIVER INJURY; LIVER ISCHEMIA; MACROPHAGE ACTIVATION; MALE; NEUTROPHIL; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT; REPERFUSION INJURY;

ADENOSINE TRIPHOSPHATE; ANIMALS; COMPLEMENT ACTIVATION; COMPLEMENT SYSTEM PROTEINS; ENDOTOXINS; GLUTATHIONE; LIVER; MACROPHAGES; MALE; NECROSIS; NEUTROPHILS; OXIDATION-REDUCTION; PROPIONIBACTERIUM ACNES; RATS; RATS, INBRED F344; SALMONELLA ENTERITIDIS; TUMOR NECROSIS FACTOR-ALPHA;

EID: 0028213842     PISSN: 02709139     EISSN: 15273350     Source Type: Journal    
DOI: 10.1002/hep.1840190424     Document Type: Article

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