Inflammation and coronary artery disease

New England Journal of Medicine

Volumn 331, Issue 7, 1994, Pages 468-469

  Alexander, R Wayne ^a  

^a EMORY UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID A PROTEIN; ANTILIPEMIC AGENT; C REACTIVE PROTEIN; CREATINE KINASE; TROPONIN T;

ATHEROSCLEROSIS; ATHEROSCLEROTIC PLAQUE; CORONARY ARTERY DISEASE; CORONARY ARTERY OBSTRUCTION; DIET RESTRICTION; EDITORIAL; HEART INFARCTION; HUMAN; INFLAMMATION; INFLAMMATORY INFILTRATE; NONHUMAN; PRIORITY JOURNAL; PROGNOSIS; UNSTABLE ANGINA PECTORIS;

AMYLOID PROTEIN AA; ANGINA, UNSTABLE; ARTERIOSCLEROSIS; C-REACTIVE PROTEIN; CHRONIC DISEASE; CORONARY DISEASE; HUMAN; INFLAMMATION; MYOCARDIAL INFARCTION;

EID: 0028087793     PISSN: 00284793     EISSN: 15334406     Source Type: Journal    
DOI: 10.1056/NEJM199408183310709     Document Type: Editorial

References (12)

1. Liuzzo G, Biasucci LM, Gallimore JR, The prognostic value of C-reactive protein and serum amyloid A protein in severe unstable angina. N Engl J Med 1994; 331: 417-424
2. Sato T, Takebayashi S, Kohchi K Increased subendothelial infiltration of the coronary arteries with monocytes/macrophages in patients with unstable angina: histological data on 14 autopsied patients. Atherosclerosis 1987; 68: 191-197
3. Munro JM, Cotran RS The pathogenesis of atherosclerosis: atherogenesis and inflammation. Lab Invest 1988; 58: 249-261
4. Ross R The pathogenesis of atherosclerosis: a perspective for the 1990s. Nature 1993; 362: 801-809
5. Stemme S, Holm J, Hansson GK T lymphocytes in human atherosclerotic plaques are memory cells expressing CD45RO and the integrin VLA-1. Arterioscler Thromb 1992; 12: 206-211
6. Alexander RW. The coronary ischemic syndromes: relationship to the biology of atherosclerosis. In: Schlant RC, Alexander RW, eds. Hurst's the heart: arteries and veins. 8th ed. New York: McGraw-Hill, 1994:1021-31.
7. van der Wal AC, Becker AE, van der Loos CM, Das PK Site of intimal rupture or erosion of thrombosed coronary atherosclerotic plaques is characterized by an inflammatory process irrespective of the dominant plaque morphology. Circulation 1994; 89: 36-44
8. Ambrose JA, Tannenbaum MA, Alexopoulos D, Angiographic progression of coronary artery disease and the development of myocardial infarction. J Am Coll Cardiol 1988; 12: 56-62
9. Little WC, Constantinescu M, Applegate RJ, Can coronary angiography predict the site of a subsequent myocardial infarction in patients with mild-to-moderate coronary artery disease? Circulation 1988; 78: 1157-1166
10. Brown G, Albers JJ, Fisher LD, Regression of coronary artery disease as a result of intensive lipid-lowering therapy in men with high levels of apolipoprotein B. N Engl J Med 1990; 323: 1289-1298
11. Harrison DG, Armstrong ML, Freiman PC, Heistad DD Restoration of endothelium-dependent relaxation by dietary treatment of atherosclerosis. J Clin Invest 1987; 80: 1808-1811
12. Stuart J, Stone PC, Akinola NO, Gallimore JR, Pepys MB Monitoring the acute phase response to vaso-occlusive crisis in sickle cell disease. J Clin Pathol 1994; 47: 166-169