An alternatively spliced form of the nerve growth factor receptor TrkA confers an enhanced response to neurotrophin 3

Proceedings of the National Academy of Sciences of the United States of America

Volumn 91, Issue 23, 1994, Pages 11133-11137

  Clary, Douglas O ^a,b   Reichardt, Louis F ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b SUGEN INC   (United States)

Author keywords

neurite outgrowth; PC12 cells

Indexed keywords

NERVE GROWTH FACTOR; NERVE GROWTH FACTOR RECEPTOR; NEUROTROPHIN 3; PROTEIN TYROSINE KINASE;

ALTERNATIVE RNA SPLICING; ANIMAL CELL; ARTICLE; AUTOPHOSPHORYLATION; CELL DIFFERENTIATION; CONTROLLED STUDY; FIBROBLAST; GENE ACTIVATION; MOLECULAR INTERACTION; NERVE FIBER GROWTH; NONHUMAN; PRIORITY JOURNAL; RAT; RECEPTOR BINDING; SIGNAL TRANSDUCTION;

ALTERNATIVE SPLICING; ANIMAL; CELL DIFFERENTIATION; GENE EXPRESSION; NERVE GROWTH FACTORS; NEUROTROPHIN 3; PC12 CELLS; PROTO-ONCOGENE PROTEINS; RATS; RECEPTOR PROTEIN-TYROSINE KINASES; RECEPTOR, TRKA; RECEPTORS, NERVE GROWTH FACTOR; RNA, MESSENGER; SIGNAL TRANSDUCTION; STRUCTURE-ACTIVITY RELATIONSHIP; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.;

ANIMALIA;

EID: 0028076182     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.91.23.11133     Document Type: Article

References (32)