Inactivation of the p53 gene by either mutation or HPV infection is extremely frequent in human oral squamous cell carcinoma cell lines

European Journal of Cancer. Part B: Oral Oncology

Volumn 30, Issue 5, 1994, Pages 338-345

  Min, Byung Moo ^a   Baek, Jeong Hwa ^a   Shin, Ki Hyuk ^a   Gujuluva, Chandrasekhar N ^a   Cherrick, Henry M ^a   Park, No Hee ^a  

^a UCLA SCHOOL OF DENTISTRY   (United States)

Author keywords

human papillomavirus; oral cancer cells; p53; tumour suppressor gene

Indexed keywords

PROTEIN P53;

ARTICLE; CONTROLLED STUDY; GENE ACTIVITY; GENE MUTATION; HUMAN; HUMAN CELL; MOUTH CARCINOMA; PAPILLOMA VIRUS; PRIORITY JOURNAL; TUMOR CELL LINE; TUMOR SUPPRESSOR GENE;

BASE SEQUENCE; BLOTTING, NORTHERN; BLOTTING, WESTERN; CARCINOMA, SQUAMOUS CELL; DNA MUTATIONAL ANALYSIS; GENE DELETION; GENES, P53; HUMAN; MOLECULAR SEQUENCE DATA; MOUTH NEOPLASMS; OPEN READING FRAMES; PAPILLOMAVIRUS, HUMAN; PAPOVAVIRIDAE INFECTIONS; POINT MUTATION; POLYMERASE CHAIN REACTION; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.; TUMOR CELLS, CULTURED; TUMOR VIRUS INFECTIONS;

EID: 0027991315     PISSN: 09641955     EISSN: None     Source Type: Journal    
DOI: 10.1016/0964-1955(94)90036-1     Document Type: Article

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