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Volumn 63, Issue 2, 1994, Pages 584-591
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Isolated Cerebral and Cerebellar Mitochondria Produce Free Radicals when Exposed to Elevated Ca2+ and Na+: Implications for Neurodegeneration
a
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Author keywords
Cerebellum; Cerebral cortex; Electron paramagnetic resonance; Excitotoxicity; Glutamate; Hydroxyl; Kainate; NMDA; Reperfusion injury; Superoxide
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Indexed keywords
GLUTA;
CALCIUM ION;
GLUTAMIC ACID;
KAINIC ACID;
N METHYL DEXTRO ASPARTIC ACID;
SODIUM ION;
ALZHEIMER DISEASE;
ANIMAL TISSUE;
ARTICLE;
BRAIN MITOCHONDRION;
CALCIUM CELL LEVEL;
CALCIUM MOBILIZATION;
ELECTRON SPIN RESONANCE;
ENZYME ACTIVATION;
HUNTINGTON CHOREA;
MALE;
NERVE CELL DEGENERATION;
NERVE CELL NECROSIS;
NERVE DEGENERATION;
NONHUMAN;
OXIDATION REDUCTION REACTION;
PARKINSON DISEASE;
PATHOPHYSIOLOGY;
PRIORITY JOURNAL;
RAT;
SODIUM CELL LEVEL;
ADENOSINE DIPHOSPHATE;
ANIMAL;
CALCIUM;
CELL HYPOXIA;
CEREBELLUM;
CEREBRAL CORTEX;
COMPARATIVE STUDY;
ELECTRON SPIN RESONANCE SPECTROSCOPY;
ELECTRON TRANSPORT;
FREE RADICALS;
HUMAN;
HYDROXYL RADICAL;
MALE;
MITOCHONDRIA;
NAD(P)H DEHYDROGENASE (QUINONE);
NERVE DEGENERATION;
NEURONS;
NITROGEN OXIDES;
PARKINSON DISEASE;
RATS;
RATS, WISTAR;
SODIUM;
SPIN LABELS;
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EID: 0027933701
PISSN: 00223042
EISSN: 14714159
Source Type: Journal
DOI: 10.1046/j.1471-4159.1994.63020584.x Document Type: Article |
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Times cited : (462)
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References (0)
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