Modulation of protein kinase C alters hemodynamics and metabolism in the isolated liver in fed and fasted rats

Journal of Hepatology

Volumn 19, Issue 3, 1993, Pages 475-484

  Inaba, Hideo ^a   Araki, Masahiko ^a   Numai, Takeshi ^a  

^a CHIBA UNIVERSITY SCHOOL OF MEDICINE   (Japan)

Author keywords

4 Phorbol; 4 Phorbol didecanoate; Glucose; Isolated liver perfusion; Lactate; Phorbol 12 myristate 13 acetate; Staurosporine

Indexed keywords

DIMETHYL SULFOXIDE; GLUCOSE; LACTIC ACID; PHORBOL 13 ACETATE 12 MYRISTATE; PHORBOL ESTER; PROTEIN KINASE C; PROTEIN KINASE INHIBITOR; STAUROSPORINE;

ANIMAL TISSUE; ARTICLE; ISOLATED LIVER; LIVER BLOOD FLOW; LIVER METABOLISM; NONHUMAN; PRIORITY JOURNAL; RAT;

ALKALOIDS; ANIMAL; ENZYME ACTIVATION; FASTING; FOOD; HEMODYNAMICS; IN VITRO; LIVER; LIVER CIRCULATION; MALE; ORGAN WEIGHT; PERFUSION; PHORBOL ESTERS; PHORBOLS; PROTEIN KINASE C; RATS; RATS, SPRAGUE-DAWLEY; STAUROSPORINE; TETRADECANOYLPHORBOL ACETATE;

EID: 0027728965     PISSN: 01688278     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0168-8278(05)80561-6     Document Type: Article

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5. Tumor necrosis factor production by Kupffer cell requires protein kinase C activation
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12. Cardiac output and its distribution in peritonitis (septic) shock in the rat
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14. Stimulation of hepatic glycogenolysis by phorbol 12-myristate 13-acetate
15. 2 mediates the stimulation of glycogenolysis in the liver by phorbol ester
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27. 2+ mediates phagocytosis-evoked eicosanoid synthesis in Kupffer cells
28. 2+ requirement of prostanoid but not of superoxide production by rat Kupffer cells