Transforming growth factor β1 null mutation in mice causes excessive inflammatory response and early death

Proceedings of the National Academy of Sciences of the United States of America

Volumn 90, Issue 2, 1993, Pages 770-774

  Kulkarni, A B ^a   Huh, C G ^a   Becker, D ^a   Geiser, A ^a   Lyght, M ^a   Flanders, K C ^a   Roberts, A B ^a   Sporn, M B ^a   Ward, J M ^a   Karlsson, S ^a  

^a NATIONAL INSTITUTES OF HEALTH   (United States)

Author keywords

autoimmunity; embryonic stem cells; homologous recombination; inflammation

Indexed keywords

DNA; RNA; TRANSFORMING GROWTH FACTOR BETA1;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AUTOIMMUNITY; CHIMERA; DEATH; DNA DETERMINATION; EMBRYO; EMBRYO CELL; FETUS; GENETIC RECOMBINATION; GRAFT VERSUS HOST REACTION; HISTOPATHOLOGY; IMMUNOREGULATION; INFLAMMATION; LYMPHOCYTE; MACROPHAGE; MOUSE; NONHUMAN; PRIORITY JOURNAL; RNA ANALYSIS; SOUTHERN BLOTTING; STEM CELL; VIRUS INFECTION;

ALLELES; ANIMAL; BASE SEQUENCE; CHIMERIC PROTEINS; EMBRYO; INFLAMMATION; LEUKEMIC INFILTRATION; LUNG; MICE; MICE, MUTANT STRAINS; MOLECULAR SEQUENCE DATA; MUTATION; MYOCARDIUM; PHENOTYPE; SYNDROME; TRANSFORMATION, GENETIC; TRANSFORMING GROWTH FACTOR BETA;

EID: 0027531528     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.90.2.770     Document Type: Article

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