Role of cytokines in the pathogenesis of cardiopulmonary-induced multisystem organ failure

The Annals of Thoracic Surgery

Volumn 56, Issue SUPPL. 5, 1993, Pages

  Casey, Larry C ^a  

^a RUSH UNIVERSITY MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

APROTININ; COMPLEMENT INHIBITOR; CYTOKINE; INTERLEUKIN 1; INTERLEUKIN 6; MONOCLONAL ANTIBODY; NAFAMSTAT; PROSTACYCLIN; THROMBOCYTE ACTIVATING FACTOR; THROMBOXANE RECEPTOR BLOCKING AGENT; TUMOR NECROSIS FACTOR;

COMPLEMENT ACTIVATION; CONFERENCE PAPER; DRUG THERAPY; ENDOTHELIUM; HUMAN; INFLAMMATION; MULTIPLE ORGAN FAILURE; PATHOGENESIS; PRIORITY JOURNAL;

ADULT; CARDIOPULMONARY BYPASS; CYTOKINES; HUMAN; MULTIPLE ORGAN FAILURE; RESPIRATORY DISTRESS SYNDROME, ADULT;

EID: 0027525711     PISSN: 00034975     EISSN: None     Source Type: Journal    
DOI: 10.1016/0003-4975(93)91143-B     Document Type: Article

References (12)

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9. Tumor necrosis factor/cachectin induction of hemorrhagic necrosis in normal tissue requires the fifth component of complement (C5)
10. Endotoxin release and tumor necrosis factor formation during cardiopulmonary bypass
11. Cytokine responses to cardiopulmonary bypass with membrane and bubble oxygenation
12. Complement activation and lung permeability during cardiopulmonary bypass