Deletion polymorphism in angiotensin-converting enzyme gene associated with parental history of myocardial infarction

The Lancet

Volumn 341, Issue 8851, 1993, Pages 991-992

  Tiret, L ^a   Nicaud, V ^a   Kee, F ^b   Evans, A ^b   Cambou, J P ^b   Arveiler, D ^b   Luc, G ^b   Amouyel, P ^b   Poirier, O ^a   Lecerf, L ^a   Cambien, F ^a  

^a INSERM   (France)

^b MONICA Projects   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

DIPEPTIDYL CARBOXYPEPTIDASE;

ADULT; ARTICLE; FEMALE; GENE LOCUS; GENETIC POLYMORPHISM; HEART INFARCTION; HEREDITY; HUMAN; MAJOR CLINICAL STUDY; MALE; PRIORITY JOURNAL; RISK FACTOR;

ADULT; FEMALE; FRANCE; GENE DELETION; HUMAN; MALE; MIDDLE AGE; MYOCARDIAL INFARCTION; NORTHERN IRELAND; ODDS RATIO; PEPTIDYL-DIPEPTIDASE A; POLYMORPHISM (GENETICS); RISK; SUPPORT, NON-U.S. GOV'T;

EID: 0027499928     PISSN: 01406736     EISSN: None     Source Type: Journal    
DOI: 10.1016/0140-6736(93)91075-W     Document Type: Article

References (6)

1. Deletion polymorphism in the gene for angiotensin-converting enzyme is a potent risk factor for myocardial infarction
2. Familial resemblance of plasma angiotensin-converting enzyme level: the Nancy study
3. An insertion/deletion polymorphism in the angiotensin I-converting enzyme gene accounting for half of the variance of serum enzyme levels
4. Evidence from combined segregation and linkage analysis that a variant of the angiotensin I-converting enzyme (ACE) gene controls plasma ACE levels
5. A case-control study of lipoprotein particles in two populations at contrasting risk for coronary heart disease
6. Structure of the angiotensin I-converting enzyme gene: two alternate promoters correspond to evolutionary steps of a duplicated gene