Suppression of experimental autoimmune encephalomyelitis by oral administration of myelin basic protein VI. Suppression of adoptively transferred disease and differential effects of oral vs. intravenous tolerization
+ suppressor cells inhibit the function of effector cells of experimental autoimmune encephalomyelitis through a mechanism involving transforming growth factor-beta
Oral tolerance to myelin basic protein and natural recovery from experimental autoimmune encephalomyelitis are associated with down-regulation of inflammatory cytokines and differential upregulation of TGF-β, IL-4 and PGE expression in the brain
Immunologic suppression after oral administration of antigen. I. Specific suppressor cells formed in rat Peyer's patches after oral administration of sheep erytrocytes and their systemic migration
Inhibition of specific immune response by feeding protein antigens. IV. Evidence for tolerance and specific active suppression of cell mediated immune response to ovalbumin
Suppressor T cells generated by oral tolerization to myelin basic protein suppress both in vitro and in vivo immune responses by the release of TGF-β following antigen specific triggering
Suppression of experimental autoimmune encephalomyelitis by oral administration of myelin basic protein. V. Hierarchy of suppression elicitated by myelin basic protein from different species
Down-regulation of the immune response to histocompatability antigens and prevention of sensitization by skin allografts by orally administered alloantigen