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Volumn 268, Issue 15, 1993, Pages 11304-11311
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Protein kinase A (PKA) still activates CFTR chloride channel after mutagenesis of all 10 PKA consensus phosphorylation sites
a a a a a a a a |
Author keywords
[No Author keywords available]
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Indexed keywords
CHLORIDE CHANNEL;
CYCLIC AMP DEPENDENT PROTEIN KINASE;
PROTEIN KINASE C;
TRANSMEMBRANE CONDUCTANCE REGULATOR;
ANIMAL CELL;
ARTICLE;
CHO CELL;
ENZYME ACTIVATION;
ENZYME BINDING;
ENZYME PHOSPHORYLATION;
GENE MUTATION;
HUMAN;
IMMUNOBLOTTING;
IMMUNOPRECIPITATION;
ION TRANSPORT;
MUTAGENESIS;
NONHUMAN;
PHOSPHORYLATION;
PRIORITY JOURNAL;
PROTEIN ANALYSIS;
SITE DIRECTED MUTAGENESIS;
AMINO ACID SEQUENCE;
ANIMAL;
BASE SEQUENCE;
CHLORIDE CHANNELS;
CHO CELLS;
CODON;
CYSTIC FIBROSIS;
CYSTIC FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR;
HAMSTERS;
HUMAN;
IODIDES;
ION CHANNELS;
KINETICS;
MEMBRANE POTENTIALS;
MEMBRANE PROTEINS;
MUTAGENESIS, SITE-DIRECTED;
PHOSPHATES;
PHOSPHORYLATION;
POLYMERASE CHAIN REACTION;
PROTEIN KINASES;
PROTEIN STRUCTURE, SECONDARY;
RECOMBINANT PROTEINS;
RESTRICTION MAPPING;
SUPPORT, NON-U.S. GOV'T;
SUPPORT, U.S. GOV'T, P.H.S.;
TRANSFECTION;
ANIMALIA;
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EID: 0027311276
PISSN: 00219258
EISSN: None
Source Type: Journal
DOI: None Document Type: Article |
Times cited : (264)
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References (0)
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