Cytokines in Kidney Disease: The Role of Transforming Growth Factor-β

American Journal of Kidney Diseases

Volumn 22, Issue 1, 1993, Pages 105-113

  Border, Wayne A ^a   Noble, Nancy A ^a  

^a UNIVERSITY OF UTAH SCHOOL OF MEDICINE   (United States)

Author keywords

Cytokines; diabetic nephropathy; extracellular matrix; glomerulonephritis; tumor necrosis factor

Indexed keywords

DECORIN; PROTEOGLYCAN; TRANSFORMING GROWTH FACTOR BETA;

CONFERENCE PAPER; DIABETIC NEPHROPATHY; DRUG EFFICACY; EXTRACELLULAR MATRIX; GLOMERULONEPHRITIS; INTERSTITIAL NEPHRITIS; KIDNEY DISEASE; NONHUMAN; PROTEIN RESTRICTION; PROTEOGLYCAN SYNTHESIS;

EID: 0027301072     PISSN: 02726386     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0272-6386(12)70175-0     Document Type: Article

References (32)

1. Autocrine secretion-10 years later
2. Peptide growth factors and inflammation, tissue repair, and cancer
3. Molecular repair, wound healing, and carcinogenesis: Tumor production a possible overhealing?
4. Tumors: Wounds that do not heal
5. Antagonists of transforming growth factor-,0: A novel approach to treatment of glomerulonephritis and prevention of glomerulosclerosis
6. Peptide growth factors are multifunctional
7. Cytokines in context
8. TGF-0 stimulation and inhibition of cell proliferation: New mechanistic insights
9. Extracellular matrix and glomerular disease
10. Transforming growth factorin disease: The dark side of tissue repair
11. Molecular and cellular biological techniques in the study of glomerular diseases
12. Transforming growth factor-,Q regulates production of proteoglycans by mesangial cells
13. Production of extracellular matrix by glomerular epithelial cells is regulated by transforming growth factor-01
14. Negative regulation of transforming growth factor-O by the proteoglycan decorin
15. Elevated expression of transforming growth factor-Q and proteoglycan production in experimental glomerulonephritis
16. Suppression of experimental glomerulonephritis by antiserum against transforming growth factor 01
17. The plasminogen activator/plasmin system
18. Enhanced production and extracellular deposition of the endothelial-type plasminogen activator inhibitor in cultured human lung fibroblasts by transforming growth factor-,6
19. Glomerular matrix accumulation is linked to inhibition of the plasmin protease system
20. Cell adhesion protein receptors as targets for transforming growth factor-O action
21. Regulation of cell adhesion receptors by transforming growth factor-0
22. Integrins
23. Diabetic kidney disease is linked to elevated expression of transforming growth factor-0
24. Pathogenesis of interstitial fibrosis in chronic purine aminonucleoside nephrosis
25. Transforming growth factor-,Q production in anti-glomerular basement membrane disease in the rabbit
26. Dietary protein restriction rapidly reduces transforming growth factor 0-1 expression in experimental glomerulonephritis
27. The transforming growth factor-os
28. Mechanistic interrelationships between two superfamilies: The steroid/retinoid receptors and transforming growth factor-0
29. Control of scarring in adult wounds by neutralizing antibody to transforming growth factor 0
30. Immunohistochemical localization of growth factors in fetal wound healing
31. Proteoglycans as modulators of growth factor activities
32. Natural inhibitor of transforming growth factor-O protects against scarring in experimental kidney disease