Tumor necrosis factor-α produces hepatocellular dysfunction despite normal cardiac output and hepatic microcirculation

American Journal of Physiology - Gastrointestinal and Liver Physiology

Volumn 265, Issue 1 28-1, 1993, Pages

  Wang, P ^a   Ayala, A ^a   Ba, Z F ^a   Zhou, M ^a   Perrin, M M ^a   Chaudry, I H ^a  

^a MICHIGAN STATE UNIVERSITY   (United States)

Author keywords

colloidal carbon perfusion; indocyanine green clearance; interleukin 6; laser Doppler flowmetry; sepsis

Indexed keywords

INTERLEUKIN 6; TUMOR NECROSIS FACTOR ALPHA;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; GLUCOSE BLOOD LEVEL; HEART FAILURE; HEART OUTPUT; HEMODYNAMICS; LASER DOPPLER FLOWMETRY; LIVER BLOOD FLOW; LIVER CELL DAMAGE; LIVER FUNCTION; MALE; MICHAELIS CONSTANT; MICROCIRCULATION; NONHUMAN; PRIORITY JOURNAL; RAT; SEPSIS;

ALANINE TRANSAMINASE; ANIMAL; ASPARTATE AMINOTRANSFERASES; BLOOD GLUCOSE; BLOOD PRESSURE; CARDIAC OUTPUT; HEART RATE; HEMODYNAMICS; INTERLEUKIN-6; LIVER; LIVER CIRCULATION; MALE; MICROCIRCULATION; RATS; RATS, SPRAGUE-DAWLEY; REFERENCE VALUES; SUPPORT, U.S. GOV'T, P.H.S.; TUMOR NECROSIS FACTOR;

EID: 0027297250     PISSN: 00029513     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (0)