Resetting of the tubuloglomerular feedback mechanism by blockade of NO‐synthase

Acta Physiologica Scandinavica

Volumn 148, Issue 3, 1993, Pages 359-360

  THORUP, C ^a   SUNDLER, F ^a   EKBLAD, E ^a   PERSSON, A E G ^a  

^a LUND UNIVERSITY   (Sweden)

Author keywords

Juxtaglomerular apparatus; macula densa cells; NO blockade; NO synthase; proximal tubular stop flow pressure; renal function; tubuloglomerular feedback

Indexed keywords

NITRIC OXIDE SYNTHASE;

ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; FEMALE; GLOMERULUS FILTRATION RATE; KIDNEY FUNCTION; NONHUMAN; PRIORITY JOURNAL; RAT;

AMINO ACID OXIDOREDUCTASES; ANIMAL; ARGININE; FEEDBACK; FEMALE; IMMUNOHISTOCHEMISTRY; KIDNEY GLOMERULUS; KIDNEY TUBULES; NITRIC-OXIDE SYNTHASE; NITROARGININE; RATS; RATS, SPRAGUE-DAWLEY; SUPPORT, NON-U.S. GOV'T;

EID: 0027260593     PISSN: 00016772     EISSN: 1365201X     Source Type: Journal    
DOI: 10.1111/j.1748-1716.1993.tb09569.x     Document Type: Article

References (3)

1. Immunohistochemical localisation of peripheral nitric oxide synthase‐containing nerves using antibodies raised against synthesized C‐ or N‐terminal fragments of cloned enzyme from rat brain
2. Interstitial pressure as a modulator of tubuloglomerular feedback control
3. Nitric oxide synthase in macula densa regulates glomerular capillary pressure