Calpain may produce a CA2+-independent form of kinase C in long-term potentiation

Biochemical and Biophysical Research Communications

Volumn 189, Issue 3, 1992, Pages 1515-1520

  Suzuki, Tatsuo ^a   Okumura Noji, Kuniko ^a   Ogura, Akihiko ^b   Tanaka, Ryo ^a   Nakamura, Kyoko ^b   Kudo, Yoshihisa ^b  

^a NAGOYA CITY UNIV MEDICAL SCHOOL   (Japan)

^b MITSUBISHI KASEI INST OF LIFE SCI   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM; CALPAIN; ENZYME INHIBITOR; LEUPEPTIN; PROTEIN KINASE C; PROTEIN KINASE INHIBITOR;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; HIPPOCAMPUS; LONG TERM POTENTIATION; NONHUMAN; PRIORITY JOURNAL; RAT;

ANIMAL; ANTIBODIES, MONOCLONAL; CALCIUM CHLORIDE; CALPAIN; ELECTRIC STIMULATION; ELECTROPHORESIS, POLYACRYLAMIDE GEL; HIPPOCAMPUS; ISOENZYMES; KINETICS; MALE; PHOSPHORYLATION; PROTEIN KINASE C; PYRAMIDAL TRACTS; RATS; RATS, WISTAR; SUBCELLULAR FRACTIONS; SUPPORT, NON-U.S. GOV'T;

ANIMALIA;

EID: 0027080526     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/0006-291X(92)90247-I     Document Type: Article

References (22)

1. Development of hippocampal long-term potentiation is reduced by recently introduced calpain inhibitors