Nitric oxide production within cardiac myocytes reduces their contractility in endotoxemia

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 263, Issue 6 32-6, 1992, Pages

  Brady, A J B ^a   Poole Wilson, P A ^a   Harding, S E ^a   Warren, J B ^a  

^a NATIONAL HEART AND LUNG INSTITUTE   (United Kingdom)

Author keywords

endotoxic shock; lipopolysaccharide; N(G) nitro L arginine methyl ester; nitric oxide synthase

Indexed keywords

ARGININE; ARGININE DERIVATIVE; DEXAMETHASONE; DEXTRO ARGININE; ESCHERICHIA COLI ENDOTOXIN; ESCHERICHIA COLI LIPOPOLYSACCHARIDE; METHYLENE BLUE; N(G) METHYLARGININE; NITRIC OXIDE; NITRIC OXIDE SYNTHASE;

ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; CELL ISOLATION; CONTROLLED STUDY; CYTOTOXICITY; ELECTROSTIMULATION; ENDOTOXEMIA; ENZYME ACTIVITY; ENZYME INHIBITION; GUINEA PIG; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILITY; HEART VENTRICLE WALL; MUSCLE FIBER CONTRACTION; NONHUMAN; PRIORITY JOURNAL; SEPTIC SHOCK;

EID: 0027053340     PISSN: 00029513     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.1992.263.6.h1963     Document Type: Article

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