TNF and IL‐1 generation by human monocytes in response to biomaterials

Journal of Biomedical Materials Research

Volumn 26, Issue 7, 1992, Pages 851-859

  Cardona, Mario A ^a   Simmons, Richard L ^a   Kaplan, Sandra S ^a  

^a UNIVERSITY OF PITTSBURGH SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BIOLOGICAL MATERIALS - CELLS; BIOMEDICAL ENGINEERING - CYTOLOGY; BIOMEDICAL ENGINEERING - SURGICAL IMPLANTS; BIOPOLYMERS - MEDICAL APPLICATIONS;

HUMAN MONOCYTES (HM); INTERLEUKIN-1 (IL-1); TUMOR NECROSIS FACTOR (TNF);

BIOMATERIALS;

BIOMATERIAL; DACRON; INTERLEUKIN 1; POLITEF; POLYSTYRENE; POLYURETHAN; TUMOR NECROSIS FACTOR;

ARTICLE; BIOCOMPATIBILITY; CELL CULTURE; HUMAN CELL; MONOCYTE;

ADULT; CELLS, CULTURED; ENZYME-LINKED IMMUNOSORBENT ASSAY; HUMAN; INTERLEUKIN-1; LIPOPOLYSACCHARIDES; MATERIALS TESTING; MONOCYTES; POLYETHYLENE TEREPHTHALATES; POLYMERS; POLYTETRAFLUOROETHYLENE; SUPPORT, U.S. GOV'T, P.H.S.; TUMOR NECROSIS FACTOR;

EID: 0026898255     PISSN: 00219304     EISSN: 10974636     Source Type: Journal    
DOI: 10.1002/jbm.820260703     Document Type: Article

References (34)

1. In vivo biocompatibility studies. V: In vivo leukocyte interactions with Biomer
2. The relationship between biocompatibility and interleukin‐1
3. The role of biomaterials science in the next generation of artificial organ technology
4. Biomaterial biocompatibility and the macrophage
5. Polymorphonuclear leukocyte degranuylation with exposure to polymethylmethacrylate nanoparticles
6. An in vivo model to study pathobiology of infectious biofilms on biomaterial surfacts
7. Mechanisms of exocytosis in phagocytic inflammatory cells
8. Macrophage fibrinolytic activity: Identification of 2 pathways of plasmin formation by intact cells and of a plasminogen activator inhibitor
9. The macrophage as a source of complement components
10. Plasma protein adsorbed biomedical polymers: Activation of human monocytes and induction of interleukin‐1
11. Generation of IL‐1 like activity in response to biomedical polymer implants: A Comparison of in vitro and in vivo models
12. Human monocyte/macrophage activation and interleukin‐1 generation by biomedical polymers
13. The detection by immunofluorescence of distinct cell populations producing interleukin‐1 in activated human peripheral bl0ood
14. Tumor necrosis factor is an endogenous pyrogen and induces jproduction of interleukin 1
15. The biology of cachectin/TNF. A primary mediatorof the hos tresponse
16. Stimulation of neutrophils by tumor necrosis factor
17. Human tumor ncrosis factor production, purification and characterization
18. The biochemistry and pharmacology of interleukins‐1 and ‐6
19. Interleukin 1 replease by human alveolar macrophages and blood monocytes
20. Cell‐associated Tumor Necrosis Factor (TNF) as a killing mechanism of activated cytotoxic macrophages
21. Tumornecrosis factor regulates expression of receptors for formyl‐methionylleucyl‐phenylalanine, leukotriene B4 and platelet activating factor. Dissociation from priming in human polymorphonuclear neutrophils
22. Release of Tumor Necrosis Factor by human peritoneal macrophages in vivo and in vitro
23. Isolation of mononuclear cells and granulocytes from human blood
24. Deetection of circulating Tumor Necrosis Factor after endotoxin administration
25. A flow cytometric and immunofluorescence microscopic study of Tumor Necrosis Factor production and localization in human monocytes
26. The role of endothelial cells in inflammation
27. Emigration of lymphocytes in rheumatoid arthritis
28. Proinflammatory cytokines, Interleukin 1 and Tumor Necrosis Factor induce cytokines that are chemotactic for neutrophils, T cells and monocytes. Cytokines and lipocortins
29. Stimulation and priming of human Neutrophils by interleukin‐;8: Cooperation with tumor necrosis factor and colony stimulating factors
30. IL‐8 causes in vivo neutrophil migration by a cell‐dependent mechanism
31. Differential production of interleukin‐1 on the surface of biomaterials
32. Human monocytes produce IL‐1 and aninhibitor of II‐1 in response to two different signals