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Volumn 70, Issue 6, 1992, Pages 923-935
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Altered cell cycle arrest and gene amplification potential accompany loss of wild-type p53
a,c a,b,c a,c a,c b a,b,c |
Author keywords
[No Author keywords available]
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Indexed keywords
PROTEIN P53;
ALLELE;
ANIMAL CELL;
ARTICLE;
CELL CYCLE;
CELL GROWTH;
CELL TRANSFORMATION;
DIPLOIDY;
FIBROBLAST;
GENE AMPLIFICATION;
GROWTH INHIBITION;
HUMAN;
HUMAN CELL;
MOUSE;
PRIORITY JOURNAL;
TUMOR SUPPRESSOR GENE;
ANIMAL;
ASPARTATE CARBAMOYLTRANSFERASE;
ASPARTIC ACID;
BASE SEQUENCE;
CARBAMOYL-PHOSPHATE SYNTHASE (GLUTAMINE-HYDROLYZING);
CELL CYCLE;
CELLS, CULTURED;
CHROMOSOME DELETION;
CYTOGENETICS;
DIHYDROOROTASE;
EMBRYO;
FIBROBLASTS;
GENE AMPLIFICATION;
GENES, P53;
GERM CELLS;
HETEROZYGOTE;
HUMAN;
LI-FRAUMENI SYNDROME;
MICE;
MICE, TRANSGENIC;
MOLECULAR SEQUENCE DATA;
MULTIENZYME COMPLEXES;
MUTATION;
PHOSPHONOACETIC ACID;
SUPPORT, NON-U.S. GOV'T;
SUPPORT, U.S. GOV'T, P.H.S.;
TUMOR CELLS, CULTURED;
ANIMALIA;
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EID: 0026801062
PISSN: 00928674
EISSN: None
Source Type: Journal
DOI: 10.1016/0092-8674(92)90243-6 Document Type: Article |
Times cited : (1281)
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References (52)
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