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Volumn 89, Issue 11, 1992, Pages 4845-4849
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Tumor necrosis factor soluble receptors circulate during experimental and clinical inflammation and can protect against excessive tumor necrosis factor α in vitro and in vivo
a a a a a a |
Author keywords
cytokine inhibitors; endotoxemia; humans; primates; septic shock
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Indexed keywords
COMPLEMENTARY DNA;
CYTOKINE;
DRUG BINDING PROTEIN;
DRUG RECEPTOR;
INTERLEUKIN 1BETA;
INTERLEUKIN 6;
RECOMBINANT PROTEIN;
TUMOR NECROSIS FACTOR ALPHA;
TUMOR NECROSIS FACTOR BINDING PROTEIN;
ARTICLE;
BABOON;
BACTEREMIA;
CLINICAL ARTICLE;
CONTROLLED STUDY;
CRITICAL ILLNESS;
CYTOTOXICITY;
DRUG INHIBITION;
ENDOTOXEMIA;
HUMAN;
HUMAN EXPERIMENT;
IMMUNOASSAY;
IMMUNOREACTIVITY;
INFLAMMATION;
MOLECULAR CLONING;
NONHUMAN;
NUCLEOTIDE SEQUENCE;
PRIORITY JOURNAL;
SEPSIS;
SEPTIC SHOCK;
ANIMAL;
CELL SURVIVAL;
HUMAN;
INFLAMMATION;
LIPOPOLYSACCHARIDES;
PAPIO;
RECEPTORS, CELL SURFACE;
RECEPTORS, TUMOR NECROSIS FACTOR;
RECOMBINANT PROTEINS;
SEPTICEMIA;
SOLUBILITY;
SUPPORT, NON-U.S. GOV'T;
SUPPORT, U.S. GOV'T, P.H.S.;
TUMOR NECROSIS FACTOR;
PAPIO HAMADRYAS;
PRIMATES;
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EID: 0026741275
PISSN: 00278424
EISSN: None
Source Type: Journal
DOI: 10.1073/pnas.89.11.4845 Document Type: Article |
Times cited : (787)
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References (0)
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