Induction of apoptosis in cultured hepatocytes and in the regressing liver by transforming growth factor-β1 occurs without activation of an endonuclease

Toxicology Letters

Volumn 64-65, Issue C, 1992, Pages 701-704

  Oberhammer, Franziska ^a   Fritsch, G ^b   Pavelka, M ^c   Froschl, G ^a   Tiefenbacher, R ^a   Purchio, T ^d   Schulte Hermann, Rolf ^a  

^a UNIVERSITY OF VIENNA   (Austria)

^b ST ANNA CHILDREN S HOSPITAL   (Austria)

^c UNIVERSITY OF INNSBRUCK   (Austria)

^d Oncogen   (United States)

Author keywords

Apoptosis; Endonuclease; TGF 1

Indexed keywords

CYPROTERONE ACETATE; ENDONUCLEASE; TRANSFORMING GROWTH FACTOR BETA1;

ANIMAL CELL; APOPTOSIS; CELL CULTURE; CONFERENCE PAPER; LIVER CELL; NONHUMAN; PRIORITY JOURNAL;

ANIMAL; APOPTOSIS; CELLS, CULTURED; DNA DAMAGE; ENDONUCLEASES; ENZYME ACTIVATION; LIVER; MICE; TRANSFORMING GROWTH FACTOR BETA;

EID: 0026690059     PISSN: 03784274     EISSN: None     Source Type: Journal    
DOI: 10.1016/0378-4274(92)90250-N     Document Type: Article

References (8)

1. Cell death: The significance of apoptosis
2. Glucocorticoid activation of a calcium-dependent endonuclease in thymocyte nuclei leads to cell death
3. Calcium-activated DNA fragmentation in rat liver nuclei
4. Expression of transforming growth factor-β in the rat ventral prostate during castration-induced programmed cell death
5. Effects of transforming growth factor-β on cell death of cultured rat hepatocytes
6. Induction of apoptosis in cultured hepatocytes and in regressing liver by transforming growth factor-β1
7. Endogenous nuclease
8. Insensitivity to the cytolytic effects of glucocorticoids in vivo is associated with a novel “slow death” phenotype