Divergent effects of propranolol on neutrophil superoxide release: Involvement of phosphatidic acid and diacylglycerol as second messengers

Biochemical and Biophysical Research Communications

Volumn 175, Issue 2, 1991, Pages 423-429

  English, Denis ^a   Taylor, Gregory S ^a  

^a INDIANA UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DIACYLGLYCEROL; PHOSPHATIDIC ACID; PROPRANOLOL; SUPEROXIDE;

ARTICLE; HUMAN; HUMAN CELL; NEUTROPHIL; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

CYTOCHALASIN B; DIGLYCERIDES; ENZYME ACTIVATION; HUMAN; IN VITRO; N-FORMYLMETHIONINE LEUCYL-PHENYLALANINE; NADH, NADPH OXIDOREDUCTASES; NADPH OXIDASE; NEUTROPHILS; PHOSPHATIDIC ACIDS; PROPRANOLOL; SIGNAL TRANSDUCTION; SUPEROXIDES; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.;

EID: 0025901149     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/0006-291X(91)91581-V     Document Type: Article

References (16)

1. Calcium influx stimulates a second pathway for sustained diacylglycerol production in leukocytes activated by chemoattractants.
2. Chemotactic factor causes rapid decreases in phosphatidylinositol,4,5-bisphosphate and phosphatidylinositol 4-monophosphate in rabbit neutrophils
3. Modulation of rat brain cytosolic phosphatidate phosphohydrolase: Effect of cationic amphiphilic drugs and divalent cations