Mutant p53 DNA clones from human colon carcinomas cooperate with ras in transforming primary rat cells: a comparison of the "hot spot" mutant phenotypes.

Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research

Volumn 1, Issue 12, 1990, Pages 571-580

  Hinds, P W ^a   Finlay, C A ^a   Quartin, R S ^a   Baker, S J ^a   Fearon, E R ^a   Vogelstein, B ^a   Levine, A J ^a  

^a PRINCETON UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

HEAT SHOCK PROTEIN; HRAS PROTEIN, HUMAN; PROTEIN P21; PROTEIN P53;

ANIMAL; ARTICLE; CELL CULTURE; CELL TRANSFORMATION; COLON TUMOR; GENETICS; HALF LIFE TIME; HUMAN; MACROMOLECULE; METABOLISM; MOLECULAR CLONING; MOLECULAR WEIGHT; MOUSE; MUTATION; NUDE MOUSE; PHYSIOLOGY; RAT; STRUCTURE ACTIVITY RELATION;

ANIMALS; CELL TRANSFORMATION, NEOPLASTIC; CELLS, CULTURED; CLONING, MOLECULAR; COLONIC NEOPLASMS; HALF-LIFE; HEAT-SHOCK PROTEINS; HUMANS; MACROMOLECULAR SUBSTANCES; MICE; MICE, NUDE; MOLECULAR WEIGHT; MUTATION; PROTO-ONCOGENE PROTEINS P21(RAS); RATS; STRUCTURE-ACTIVITY RELATIONSHIP; TUMOR SUPPRESSOR PROTEIN P53;

EID: 0025618980     PISSN: 10449523     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (0)