Flufenamic acid, mefenamic acid and niflumic acid inhibit single nonselective cation channels in the rat exocrine pancreas

FEBS Letters

Volumn 268, Issue 1, 1990, Pages 79-82

  Gogelein H ^a   Dahlem, D ^a   Englert, H C ^a   Lang, H J ^a  

^a MAX PLANCK INSTITUTE OF BIOPHYSICS   (Germany)

Author keywords

Flufenamic acid; Indomethacin; Mefenamic acid; Niflumic acid; Non selective cation channel; Rat exocrine pancreas

Indexed keywords

ACETYLSALICYLIC ACID; CATION CHANNEL; DILTIAZEM; FLUFENAMIC ACID; IBUPROFEN; INDOMETACIN; MEFENAMIC ACID; NIFLUMIC ACID;

ANIMAL CELL; ARTICLE; NONHUMAN; PANCREAS; PRIORITY JOURNAL; RAT;

ANIMAL; ANTI-INFLAMMATORY AGENTS, NON-STEROIDAL; CATIONS; ELECTRIC CONDUCTIVITY; FLUFENAMIC ACID; IN VITRO; ION CHANNELS; MEFENAMIC ACID; NICOTINIC ACIDS; NIFLUMIC ACID; PANCREAS; RATS;

ANIMALIA;

EID: 0025372566     PISSN: 00145793     EISSN: None     Source Type: Journal    
DOI: 10.1016/0014-5793(90)80977-Q     Document Type: Article

References (23)

1. The nonselective cation channel in the basolateral membrane of rat exocrine pancreas
2. A voltage-dependent ionic channel in the basolateral membrane of late proximal tubules of the rabbit kidney
3. Patch-clamp study of single-channel and whole-cell K+ currents in guinea pig pancreatic acinar cells.
4. Single potassium channels blocked by lidocaine and quinidine in isolated turtle colon epithelial cells.
5. Quinidine-induced inhibition of transient outward current in cardiac muscle.
6. Potassium channels in the basolateral membrane of the rectal gland ofSqualus acanthias