Induction of cytogenetic adaptive response of somatic and germ cells in vivo and in vitro by low-dose x-irradiation

International Journal of Radiation Biology

Volumn 58, Issue 1, 1990, Pages 187-194

  Cai, L ^a   Liu, S Z ^a  

^a JILIN UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

ADAPTATION; ANIMAL CELL; ARTICLE; BONE MARROW CELL; CELL CULTURE; CYTOGENETICS; CYTOLOGY; GERM CELL; HUMAN; HUMAN CELL; LOW ENERGY RADIATION; LYMPHOCYTE; PRIORITY JOURNAL; RABBIT; X IRRADIATION;

ADAPTATION, BIOLOGICAL; ANIMAL; BONE MARROW; BONE MARROW CELLS; CELL CYCLE; CHROMOSOME ABERRATIONS; HUMAN; IN VITRO; LYMPHOCYTES; MALE; MICE; MICE, INBRED C57BL; RABBITS; RADIATION GENETICS; SPERMATOCYTES;

ANIMALIA; ORYCTOLAGUS CUNICULUS;

EID: 0025309180     PISSN: 09553002     EISSN: None     Source Type: Journal    
DOI: 10.1080/09553009014551541     Document Type: Article

References (10)

1. A new approach in the study of the differential radiosensitivity of mammalian chromosomes
2. Chromosomal responses to ionizing radiation reminiscent of an adaptive response in cultured Chinese hamster cells
3. Adaptive response of human lymphocytes to low concentrations of radioactive thymidine
4. Exposure of human lymphocytes to ionizing radiation reduces mutagenesis by subsequent ionizing radiation
5. Very low doses of X-rays can cause human lymphocytes to become less susceptible to ionizing radiation
6. Characterization of the adaptive response to ionizing radiation induced by low doses of X-rays to human lymphocytes
7. UDS and SCE in lymphocytes of persons occupationally exposed to low levels of ionizing radiation
8. Resistance and cross-resistance to chromosome damage in human blood lymphocytes adapted to bleomycin
9. Evidence that the [3H]thymidine-induced adaptive response of human lymphocytes to subsequent doses of X-rays involves the induction of a chromosomal repair mechanism
10. Human lymphocytes exposed to low doses of ionizing radiations become refractory to high doses of radiation as well as to chemical mutagens that induce double-strand breaks in DNA