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Volumn 63, Issue 6, 1989, Pages 2644-2649
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A promotor of Epstein-Barr virus that can function during latent infection can be transactivated by EBNA-1, a viral protein required for viral DNA replication during latent infection
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Author keywords
[No Author keywords available]
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Indexed keywords
VIRUS PROTEIN;
EPSTEIN BARR VIRUS ANTIGEN;
EPSTEIN BARR VIRUS NUCLEAR ANTIGENS;
VIRUS ANTIGEN;
VIRUS DNA;
AUTORADIOGRAPHY;
CELL CULTURE;
ELECTROPORATION;
EPSTEIN BARR VIRUS;
GENETIC ENGINEERING;
IMMUNOBLOTTING;
LATENT VIRUS INFECTION;
NONHUMAN;
PRIORITY JOURNAL;
PROMOTER REGION;
TRANSACTIVATION;
VIRUS REPLICATION;
ARTICLE;
BIOSYNTHESIS;
CELL LINE;
DNA REPLICATION;
GENE EXPRESSION REGULATION;
GENETICS;
HUMAN;
IMMUNOLOGY;
PHYSIOLOGY;
PLASMID;
RESTRICTION MAPPING;
WESTERN BLOTTING;
ANTIGENS, VIRAL;
BLOTTING, WESTERN;
CELL LINE;
DNA REPLICATION;
DNA, VIRAL;
EPSTEIN-BARR VIRUS NUCLEAR ANTIGENS;
GENE EXPRESSION REGULATION;
HERPESVIRUS 4, HUMAN;
HUMAN;
PLASMIDS;
PROMOTER REGIONS (GENETICS);
RESTRICTION MAPPING;
SUPPORT, U.S. GOV'T, P.H.S.;
VIRUS REPLICATION;
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EID: 0024474724
PISSN: 0022538X
EISSN: None
Source Type: Journal
DOI: 10.1128/jvi.63.6.2644-2649.1989 Document Type: Article |
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Times cited : (185)
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References (0)
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