α 1‐Adrenergic effects and liver regeneration

Hepatology

Volumn 7, Issue 6, 1987, Pages 1189-1194

  Cruise, Jennifer L ^a   Knechtle, Stuart J ^a   Bollinger, R Randal ^a   Kuhn, Cynthia ^a   Michalopoulos, George ^a  

^a DUKE UNIVERSITY MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CATECHOLAMINE; EPIDERMAL GROWTH FACTOR; EPIDERMAL GROWTH FACTOR I 125; GUANETHIDINE; PRAZOSIN; THYMIDINE H 3; UNCLASSIFIED DRUG;

ANIMAL CELL; LIVER REGENERATION; NONHUMAN;

ANIMAL; DENERVATION; DNA; FEMALE; GUANETHIDINE; HEPATECTOMY; LIVER; LIVER REGENERATION; PRAZOSIN; RATS; RECEPTOR, EPIDERMAL GROWTH FACTOR; RECEPTORS, ADRENERGIC, ALPHA; SUPPORT, U.S. GOV'T, NON-P.H.S.; SUPPORT, U.S. GOV'T, P.H.S.;

EID: 0023579843     PISSN: 02709139     EISSN: 15273350     Source Type: Journal    
DOI: 10.1002/hep.1840070604     Document Type: Article

References (36)

1. Control of hepatocyte replication by two serum factors
2. Hormonal factors and liver growth
3. Aspects of hormonal influences on liver growth
4. Alteration in binding of dexamethasone to glucocorticoid receptors in regenerating rat liver after partial hepatectomy
5. Adrenergic agents as possible regulators of liver regeneration
6. Changes in plasma glucose and insulin levels induced by adrenergic agents in normal and alloxan‐diabetic rats
7. Induction of liver cell proliferation in intact rats by amines and glucagon
8. The hepatic adrenergic receptors
9. Epidermal growth factor: effects of androgens and adrenergic agents
10. Adrenergic effects on exocrine secretion of rat submandibular epidermal growth factor
11. Adrenergic activation of triiodothyronine production in brown adipose tissue
12. Hormonal control of liver regeneration
13. Adrenergic antagonists and a possible link between the increase in cAMP and DNA synthesis during liver regeneration
14. The effects of drugs or denervation on thymidine uptake into rat regenerating liver
15. Induction of DNA synthesis in cultured rat hepatocytes through stimulation of alpha‐1 adrenoreceptor by norepinephrine
16. Norepinephrine and epidermal growth factor: dynamics of their interaction in the stimulation of hepatocyte DNA synthesis
17. Norepinephrine decreases EGF binding in primary rat hepatocyte cultures
18. Hormonal regulation of DNA synthesis in primary cultures of adult rat hepatocytes. Action of glucagon
19. Data regarding the mechanisms of nervous regulation in the regenerating liver
20. Depressed synthesis of DNA in regenerating rat liver after spinal cord (C7) transection
21. Le modificazioni dei fenomeni primari della rigenerazione epatica sotto l'influenza di alcuni sostanze stimolanti e di vari interventi chirurgici practicati sul sistema nervoso centrale
22. A microsurgical method for denervation of the liver in the rat
23. Decreased synthesis of DNA in regenerating rat liver after administration of reserpine
24. A possible role for norepinephrine in the control of liver regeneration
25. Guanethidine‐induced destruction of sympathetic neurons
26. Epidermal growth factor receptor number decreases during rat liver regeneration
27. Epidermal growth factor and proliferation in rat hepatocytes in primary culture isolated at different times after partial hepatectomy
28. Experimental pathology of the liver. I. Restoration of the liver of the white rat following partial surgical removal
29. An improved fluorometric assay for DNA
30. Quantitation of plasma cate‐cholamines by on‐line trace enrichment high performance liquid chromatography with electrochemical detection
31. 2+ transport by rat liver plasma membrane vesicles
32. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein‐dye binding
33. Liver regeneration studies with rat hepatocytes in primary culture
34. A vasopressin‐like peptide in the mammalian sympathetic nervous system
35. Vasopressin inhibition of epidermal growth factor binding to cultured mouse cells
36. Reduction of epidermal growth factor receptor affinity by heterologous ligands: evidence for a mechanism involving the breakdown of phosphoinositides and the activation of protein kinase C