A hemodynamic load in vivo induces cardiac expression of the cellular oncogene, c-myc

Biochemical and Biophysical Research Communications

Volumn 147, Issue 2, 1987, Pages 627-636

  Mulvagh, Sharon L ^a   Michael, Lloyd H ^a   Perryman, M Benjamin ^a   Roberts, Robert ^a   Schneider, Michael D ^a  

^a BAYLOR COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CREATINE KINASE; MESSENGER RNA;

ANIMAL CELL; ANIMAL EXPERIMENT; CONTROLLED STUDY; HEART ATRIUM; HEART VENTRICLE; HEART VENTRICLE HYPERTROPHY; NONHUMAN; ONCOGENE C MYC;

AGING; ANIMAL; AORTA; CARDIOMEGALY; COMPARATIVE STUDY; CONSTRICTION; CREATINE KINASE; GENE EXPRESSION REGULATION; HEART ATRIUM; HEART VENTRICLE; MALE; MYOCARDIUM; ONCOGENES; RATS; RATS, INBRED STRAINS; RNA, MESSENGER; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.;

EID: 0023473339     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/0006-291X(87)90977-6     Document Type: Article

References (45)

1. Cardiac myocyte hypertrophy is associated with c-myc protooncogene expression.
2. Inhibition of myogenic differentiation by fibroblast growth factor or type beta transforming growth factor does not require persistent c-myc expression.