13 Seaweed (Lyngbya majuscula) dermatitis

Clinics in Dermatology

Volumn 5, Issue 3, 1987, Pages 92-100

  Izumi, Allan K ^a,b   Moore, Richard E ^a,b  

^a UNIVERSITY OF HAWAII   (United States)

^b UNIVERSITY OF HAWAII   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CARCINOGEN; LYNGBYATOXIN; SEA WATER;

CONTACT DERMATITIS; DIFFERENTIAL DIAGNOSIS; HUMAN; REVIEW; SEAWEED; SWIMMING; UNITED STATES;

CARCINOGENS; DERMATITIS, CONTACT; DIAGNOSIS, DIFFERENTIAL; HAWAII; HUMANS; LYNGBYA TOXINS; SEAWATER; SEAWEED; SWIMMING;

EID: 0023377067     PISSN: 0738081x     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0738-081X(87)80014-7     Document Type: Article

References (49)

1. Seaweed dermatitis apparently caused by a marine alga. Clinical investigative procedures
2. Seaweed dermatitis apparently caused by a marine alga. Laboratory observations
3. Seaweed dermatitis apparently caused by a marine alga. Clinical observations
4. Seaweed dermatitis: first report of a dermatitis-producing marine alga
5. Seaweed itch on windward Oahu
6. A dermatitis-producing alga in Hawaii-preliminary report
7. Dermatitis-producing alga Lyngbya majuscula Gomont in Hawaii. I. Isolation and chemical characterization of the toxic factor
8. Dermatitis-producing alga Lyngbya majuscula Gomont in Hawaii. II. Biological properties of the toxic factor
9. Anti-leukemia activity in the oscillatoriaceae: isolation of debromoaplysiatoxin from Lyngbya
10. Dermatitis from purified sea algae toxin (debromoaplysiatoxin)
11. Seaward dermatitis: structure of lyngbyatoxin A
12. Indole alkaloids: dihydroteleocidin B, teleocidin, and lyngbyatoxin A as members of a new class of tumor promoters
13. Escharotic stomatitis caused by the “stinging seaweed” Microcoleus lyngbyaceus (formerly Lyngbya majuscula): a case report and review of literature
14. Irritant diterpene ester promoters of mouse skin: contributions to etiologies of environmental cancer and to biochemical mechanisms of carcinogenesis
15. 3H) debromoaplysiatoxin to the phorbol ester receptor in a mouse epidermal particulate fraction
16. Observations on ciguatera-type toxin in fish
17. Aplysiatoxin and debromoaplysiatoxin, constituents of the marine mollusk Stylocheilus longicauda (Quoy and Gaimard, 1824)
18. The aplysiatoxins
19. Observations on development, larval growth, and metamorphosis of four species of Aplysidae (Gastropoda: Opisthobranchia) in laboratory culture
20. Occurrence of a toxic blue-green alga inducing skin dermatitis in Okinawa
21. A blue-green alga from Okinawa contains aplysiatoxins, the third class of tumor promoters
22. The aplysiatoxins: reactions with acids and oxidants
23. Toxins, anticancer agents, and tumor promoters from marine prokaryotes
24. Absolute stereochemistries of the aplysiatoxins and oscillatoxin A
25. The structure of teleocidin B
26. X-ray structure determination of dihydroteleocidin B monobromoacetate
27. A new toxic substance, teleocidin, produced by streptomyces. Part IV. Degradative studies of hydroteleocidin B and teleocidic anhydride
28. Similarity of teleocidin B and phorbol ester tumor promoters in effects on membrane receptors
29. Teleocidin from streptomyces is a potent promoter of mouse skin carcinogenesis
30. A two-stage mouse skin carcinogenesis study of lyngbyatoxin A
31. New classes of tumor promoters: teleocidin, aplysiatoxin, and palytoxin
32. Synthesis of optically active teleocidin derivatives. Absolute configuration of teleocidin B and olivoretin A.
33. Absolute configuration of lyngbyatoxin A (teleocidin A-1) and teleocidin A-2
34. The third class of new tumor promoters, polyacetates (debromoaplysiatoxin and aplysiatoxin), can differentiate biological actions relevant to tumor promoters
35. Stimulation of prostaglandin production and choline turnover in HeLa cells by lyngbyatoxin A and dihydroteleocidin B
36. Comparative effects of aplysiatoxin, dibromoaplysiatoxin, and teleocidin on receptor binding and phospholipid metabolism
37. Stimulation of human polymorphonuclear leukocyte superoxide anion radical production by tumor promoters
38. Activation of the EBV-cycle and aggregation of human blood lymphocytes by the tumor promoters teleocidin, lyngbyatoxin A, aplysiatoxin and debromoaplysiatoxin
39. Teleocidin, lyngbyatoxin A and their hydrogenated derivatives, possible tumor promoters, induce terminal differentiation in HL-60 cells
40. Effects of teleocidin and the phorbol ester tumor promoters on cell transformation, differentiation, and phospholipid metabolism
41. Identification of a calcium- and phospholipid-dependent phorbol ester binding activity in the soluble fraction of mouse tissues
42. 3H)phorbol-12,13-dibutryrate binding by mouse skin
43. Tissue and species distribution and developmental variation of specific receptors for biologically active phorbol and ingenol esters
44. Direct activation of calcium-activated, phospholipid-dependent protein kinase by tumor promoting phorbol esters
45. Activation of caclium-activated, phospholipid-dependent protein kinase (protein kinase C) by new classes of tumor promoters: teleocidin and debromoaplysiatoxin
46. Activation of protein kinase C by tumor promoting phorbol esters, teleocidin and aplysiatoxin in the absence of added calcium
47. The phorbol ester receptor: a phospholipid-regulated protein kinase