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Volumn 383, Issue 1, 1987, Pages 639-652
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Non‐cholinergic component of rat splanchnic nerves predominates at low neuronal activity and is eliminated by naloxone.
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Author keywords
[No Author keywords available]
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Indexed keywords
ACETYLCHOLINE;
ATROPINE;
CATECHOLAMINE;
MECAMYLAMINE;
NALOXONE;
TETRYLAMMONIUM;
CATECHOLAMINE DERIVATIVE;
MUSCARINIC RECEPTOR;
NICOTINIC RECEPTOR;
ANIMAL CELL;
AUTONOMIC NERVOUS SYSTEM;
BIOLOGICAL MODEL;
CATECHOLAMINE RELEASE;
DOSE RESPONSE;
DRUG COMPARISON;
DRUG EFFICACY;
DRUG INHIBITION;
DRUG INTERACTION;
DRUG RESPONSE;
ENDOCRINE SYSTEM;
ETIOLOGY;
METHODOLOGY;
NERVE CELL;
NERVOUS SYSTEM;
NONCHOLINERGIC SYSTEM;
NONHUMAN;
PERIPHERAL NERVOUS SYSTEM;
PHARMACOKINETICS;
PRIORITY JOURNAL;
RAT;
REGIONAL PERFUSION;
SPLANCHNIC NERVE;
ADRENAL GLAND;
ANIMAL;
ARTICLE;
DRUG EFFECT;
ELECTROSTIMULATION;
INNERVATION;
MALE;
PHYSIOLOGY;
ACETYLCHOLINE;
ADRENAL GLANDS;
ANIMAL;
ATROPINE;
CATECHOLAMINES;
DOSE-RESPONSE RELATIONSHIP, DRUG;
ELECTRIC STIMULATION;
MALE;
MECAMYLAMINE;
NALOXONE;
RATS;
RECEPTORS, MUSCARINIC;
RECEPTORS, NICOTINIC;
SPLANCHNIC NERVES;
SUPPORT, U.S. GOV'T, NON-P.H.S.;
SUPPORT, U.S. GOV'T, P.H.S.;
TETRAETHYLAMMONIUM;
TETRAETHYLAMMONIUM COMPOUNDS;
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EID: 0023162206
PISSN: 00223751
EISSN: 14697793
Source Type: Journal
DOI: 10.1113/jphysiol.1987.sp016434 Document Type: Article |
Times cited : (56)
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References (0)
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