Post-treatments with sodium arsenite during G2 enhance the frequency of chromosomal aberrations induced by S-dependent clastogens

Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis

Volumn 163, Issue 3, 1986, Pages 263-269

  Lee, T C ^a   Tzeng, S F ^a   Chang, W J ^a   Lin, Y C ^a   Jan, K Y ^a  

^a ACADEMIA SINICA   (Taiwan)

Author keywords

[No Author keywords available]

Indexed keywords

ARSENITE SODIUM;

ANIMAL CELL; CELL CYCLE; CHO CELL; CHROMOSOME ABERRATION; CHROMOSOME BREAKAGE; ECOLOGY; FEMALE GENITAL SYSTEM; HEREDITY; HISTOLOGY; IN VITRO STUDY; MUTAGENESIS; NONHUMAN; PRIORITY JOURNAL;

ANIMALS; ARSENIC; ARSENITES; CELL LINE; CHROMATIDS; CHROMOSOME ABERRATIONS; CRICETINAE; DNA REPAIR; DRUG SYNERGISM; FEMALE; INTERPHASE; MUTAGENS; OVARY; SODIUM COMPOUNDS; ULTRAVIOLET RAYS;

EID: 0023007739     PISSN: 00275107     EISSN: None     Source Type: Journal    
DOI: 10.1016/0027-5107(86)90024-2     Document Type: Article

References (12)

1. International Workshop on Inhibition of DNA Repair
2. Effects of sodium arsenite on single-strand DNA break formation and post-replication repair in E. coli following UV irradiation
3. DNA lysis is involved in the simplified fluorescence plus Giemsa method for differential staining of sister chromatids
4. Different action modes of sodium arsenite, 3-aminobenzamide, and caffeine on the enhancement of ethyl methanesulfonate clastogenicity
5. Synergistic enhancement of the frequency of chromatid aberrations in cultured human lymphocytes by combinations of inhibitors of DNA repair
6. Sodium arsenite enhances the cytotoxicity, clastogenicity, and 6-thioguanine-resistant mutagenicity of ultraviolet light in Chinese hamster ovary cells
7. Sodium arsenite potentiates the clastogenicity and mutagenicity of DNA crosslinking agents
8. Differential effects of the pre- and post-treatment of sodium arsenite on the genotoxicity of methyl methanesulfonate in Chinese hamster ovary cells
9. Influence of inhibition of repair enzymes on the induction of chromosomal aberrations by physical and chemical agents
10. Formation of chromatid-type aberrations in G2 stage of the cell cycle
11. The use of inhibitors of DNA repair in the study of the mechanisms of induction of chromosome aberrations
12. Defective DNA repair and increased lethality in ataxia telangiectasia cells exposed to 4-nitroquinoline 1-oxide