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Teratogenesis, Carcinogenesis, and Mutagenesis

Volumn 5, Issue 3, 1985, Pages 149-158

Teratogen metabolism: Spontaneous decay products of thalidomide and thalidomide analogues are not bioactivated by liver microsomes

(2) Braun, Andrew G a Weinreb, Steven L a

a MASSACHUSETTS INSTITUTE OF TECHNOLOGY (United States)

Author keywords

hydrolysis; metabolism; thalidomide

Indexed keywords

2 (2,6 DIOXO 3 PIPERIDINYL)PHTHALIMIDINE; SUPIDIMIDE; TERATOGENIC AGENT; THALIDOMIDE; UNCLASSIFIED DRUG;

2 (2,6 DIOXOPIPERIDEN 3' YL)PHTHALIMIDINE; CELL ADHESION; CHEMICAL TERATOGENESIS; CONGENITAL DISORDER; DRUG METABOLISM; ENDOCRINE SYSTEM; FEMALE GENITAL SYSTEM; HYDROLYSIS; IN VITRO STUDY; LIVER; LIVER MICROSOME; METABOLIC ACTIVATION; MOUSE; NONHUMAN; OVARY TUMOR; SUPIDIMIDE;

ANIMAL; BIOTRANSFORMATION; CELL LINE; DRUG STABILITY; FEMALE; HYDROGEN-ION CONCENTRATION; HYDROLYSIS; KINETICS; MICE; MICE, INBRED C3H; MICROSOMES, LIVER; OVARIAN NEOPLASMS; SUPPORT, U.S. GOV'T, NON-P.H.S.; THALIDOMIDE;

EID: 0022259444 PISSN: 02703211 EISSN: 15206866 Source Type: Journal
DOI: 10.1002/tcm.1770050304 Document Type: Article

Times cited : (21)

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