Non-quantal release of transmitter at mouse neuromuscular junction and its dependence on the activity of Na+-K+ ATP-ase

Pflügers Archiv European Journal of Physiology

Volumn 370, Issue 3, 1977, Pages 295-297

  Vyskočil, František ^a   Illés, Peter ^b  

^a INSTITUTE OF MATHEMATICS   (Czech Republic)

^b SEMMELWEIS UNIVERSITY OF MEDICINE   (Hungary)

Author keywords

End plate; Mouse diaphragm; Release of ACh

Indexed keywords

ACETYLCHOLINE; ADENOSINE TRIPHOSPHATASE; NEUROTRANSMITTER; OUABAIN; TUBOCURARINE CHLORIDE;

DIAPHRAGM; MOUSE; NERVE ENDING; NEUROMUSCULAR SYNAPSE; THEORETICAL STUDY;

ACETYLCHOLINE; ADENOSINETRIPHOSPHATASE; ANIMAL; DIAPHRAGM; MEMBRANE POTENTIALS; MICE; MOTOR ENDPLATE; NEUROMUSCULAR JUNCTION; OUABAIN; POTASSIUM; SODIUM; TUBOCURARINE;

EID: 0017703013     PISSN: 00316768     EISSN: 14322013     Source Type: Journal    
DOI: 10.1007/BF00585542     Document Type: Article

References (6)

1. An investigation of spontaneous activity at the neuromuscular junction of the rat
2. Transmitter leakage from motor nerve endings
3. The mechanism of acetylcholine release from parasympathetic nerves
4. Stimulation, by inhibition of /Na+−K+−Mg++/activated ATP-ase, of acetylcholine release in cortical slices from rat brain
5. Possible connection between the release of acetylcholine and the activity of Na+−K+-activated ATP-ase
6. Termination of transmitter release by stimulation of sodium-potassium activated ATPase