Risk of cervical cancer and geographical variations of human papillomavirus 16 E6 polymorphisms

Lancet

Volumn 352, Issue 9138, 1998, Pages 1441-1442

  Zehbe, Ingeborg ^b   Voglino, Gianfranco ^b   Delius, Hajo ^b   Wilander, Erik ^b   Tommasino, Massimo ^a  

^a GERMAN CANCER RESEARCH CENTER DKFZ   (Germany)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

ARTICLE; CARCINOGENICITY; FEMALE; GENETIC POLYMORPHISM; GEOGRAPHIC DISTRIBUTION; HUMAN; ITALY; PRIORITY JOURNAL; RISK FACTOR; SWEDEN; UTERINE CERVIX CANCER; UTERINE CERVIX CARCINOMA IN SITU; WART VIRUS; WART VIRUS 16 E6;

EID: 0012652504     PISSN: 01406736     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0140-6736(05)61263-9     Document Type: Article

References (4)

1. 1 Zur Hausen H. Viruses in human cancers. Science 1991; 254: 1167-73.
2. 2 Schiffman MH, Bauer HM, Hoover RN, et al. Epidemiologic evidence showing that human papillomavirus infection causes most cervical intraepithelial neoplasia. J Natl Cancer Inst 1993; 85: 958-64.
3. 3 Londesborough O, Ho L, Terry G, et al. Human papillomavirus genotype as a predictor of persistence and development of high-grade lesions in women with minor cervical abnormalities. Int J Cancer 1996; 69: 364-68.
4. 4 Zehbe I, Wilander E, Delius H, Tommasino M. Human papillomavirus 16 E6 variants are more prevalent in invasive cervical carcinoma than the prototype. Cancer Res 1998; 58: 829-33.